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Studies indicate improvements in liver function markers and reductions in liver inflammation and fibrosis, although the precise mechanisms are still under investigation

(1996) Direct interaction between protein kinase C theta (PKC theta) and 14-3-3 tau in T cells: 14-3-3 overexpression results in inhibition of PKC theta translocation and function

However, during dysbiosis, the abnormal proliferation of pathogenic bacteria not only directly triggers the excessive release of pro-inflammatory factors but also exacerbates the hosts iron metabolism imbalance

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