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Hence, increased DA signaling might contribute to mPFC hyperexcitability as reported here and previously reported hyperactive DA neurons in the ventral tegmental area [4, 5]

Conclusions As research on ROSs has advanced, an increasing number of diseases have been found to be linked to oxidative stress

Similarly, in patients with carnitine-acylcarnitine translocase deficiency (CACTD), the carnitine shuttle system does not function and LCFAs cannot be transported inside the mitochondrial matrix for beta oxidation

[DOI] [PMC free article] [PubMed] [Google Scholar] 17.Klein S.A., Dobmeyer J.M., Dobmeyer T.S., Pape M., Ottmann O.G., Helm E.B., Hoelzer D., Rossol R