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European Journal of Cell Biology , 95 (11), 449464

This intersection positions NETosis not merely as an antimicrobial strategy but as a cytotoxic effector mechanism driving sterile inflammation, endothelial dysfunction, and thrombo-inflammatory propagation in PCOS and related metabolic disorders

Moreover, through the crosstalk with GPCR, insulin activates yes associated protein (YAP) and cMyc to mediate the altered glycolysis induced by KRAS mutation (Hao et al., 2017