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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROS–cytochrome c–caspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

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According to Li et al

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROScytochrome ccaspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROScytochrome ccaspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROScytochrome ccaspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

Long-term metformin use and nitrous oxide exposure are also recognised risk factors for B12 deficiency

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROScytochrome ccaspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model
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