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It has been shown that GDNF administration by catheter increases dopaminergic neuronal resistance against 6-OHDA toxicity, but with preservation of motor functions in rat and rhesus monkey models [96]

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The ability to predict compound concentrations and exposure durations that could cause a significant compromise in cellular antioxidant defence as a function of a physicochemical property, especially with respect to an understated mechanism such as redox cycling, could prove to be extremely useful when investigating toxicity with the reduction of animal models in mind

These groups might not get enough B12 because of their diet, age, or health issues