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Studies in pre-clinical metabolic research models use the combination to examine the body composition and metabolic consequences of pharmacologically amplified GH pulsatility characterising GH-dependent lipolysis, lean mass accretion, and insulin sensitivity changes in response to secretagogue-driven GH axis stimulation

These modifications can also be seen in the disruption of normal gene expression (85, 90)

The Ominous Octet: Multiple Pathophysiologic Defects in Type 2 Diabetes Mellitus (T2DM) 2 This review will focus on incretin-based therapies (DPP-4 inhibitors and GLP-1 receptor agonists) and SGLT-2 inhibitors currently approved for the treatment of T2DM