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(Li et al., 2022), identified that CB1 antagonists regulated sensitivity of TNBC cells to ferroptosis by regulating SCD1-and FADS2-dependent fatty acid metabolism

To more precisely examine the extent to which mitochondrial GSH depletion impacts erythropoiesis, we additionally generated a mouse model in which mitochondrial GSH import is disrupted specifically in the erythroid lineage (conditional SLC25a39-KO mice, ErGFP-cre

Cell were then treated with Cy5.5-labeled NP4 for 1 h, 4 h, or 7 h, respectively, washed with PBS, and analyzed by flow cytometry (Beckman Coulter, U.S.A.)

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