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Finally, expanding the scope of CRISPR-based screens in patient-derived xenografts (PDX) or 3D bioprinted tumor constructs could enhance the discovery of novel drug targets and resistance mechanisms, ultimately leading to more personalized and adaptive cancer therapies

Previous studies have demonstrated that GLUD1 provides a metabolic advantage to cancer cells through regulating carbon and nitrogen fluxes [2]

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elegans , elevated GLP-1/Notch activity promotes the formation of germline tumors (Berry et al., 1997)