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Mitochondrial dysfunction in ASD is further supported by the presence of non-specific biomarkers including reduction in carnitine and increases in lactate, pyruvate, creatine kinase, aspartate aminotransferase and alanine aminotransferase compared to control groups (Frye, 2020

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We found no indications of an altered placental mitochondrial content between any of the three PE subclasses and all control groups when using citrate synthase expression and mitochondrial complex IV (COX-IV) activity assays as surrogates of the mitochondrial content (40, 41) (Fig S1B and C)

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