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Current guidelines recommend baseline and periodic monitoring of liver enzymes, lipid profiles, and cholestatic markers (Sauder et al

Appetite Effects The question about appetite is interesting because the proposed mechanism could reasonably go either direction: Reduced lipogenesis and improved metabolic flexibility could produce secondary effects on appetite regulation NAD+ restoration has complex effects on hypothalamic appetite signaling Preclinical data do not consistently describe dramatic appetite changes In human use were it to become clinically available patient-reported appetite effects would be a relevant monitoring parameter, but the data do not currently support a strong a priori expectation

Deposit caps, time reminders, cooling-off periods, and self-exclusion options are prominently placed and easy to activate