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TGF- is secreted by AECs, endothelial cells, fibroblasts, myofibroblasts, and various immune cells, and it promotes EMT, metabolic reprogramming, and excessive ECM productionhallmark features of fibrosis [36, 37]

In this trial, at 60 weeks, the exenatide group showed a significant improvement in MDS-UPDRS Part III scores compared to the placebo group among the 62 recruited patients (80)

Secondary resistance in patients who showed an initial response has actually been thought to be due to de novo mutations located on regions in smoothened to which hedgehog pathway inhibitors bind or selective clonal expansion of minority clones in the pre-treated tumor

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