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A nonexclusive alternative explanation for the kinetic patterns with HEDS (and the decreased enzyme activity at high GSH concentrations) could be a Grx(SSCH 2 CH 2 OH) intermediate that is attacked by GSH according to monothiol mechanism (i) from Fig
It has gained considerable interest due to its antioxidant properties and its role in regulating blood lipid levels (147), hypertension (148), and blood glucose levels (149), among other pharmacological effects
So researchers at Washington State University chemically modified it into something sturdier that could survive being swallowed and cross the blood-brain barrier (the filter that keeps most substances in your bloodstream out of your brain tissue)
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