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GHK-Cu has demonstrated the ability to suppress excessive inflammatory signaling and reduce leukocyte infiltration in tissue injury models, indicating regulatory effects on immune-cell migration and local inflammatory cell density (31,32)

demonstrated that LPS stimulation activates ferroptosis, as evidenced by elevated levels of multiple ferroptosis markers 24 hours post-exposure [67]

found that Exos from hemeified bone marrow MSCs (HO-1/BMSCs) effectively reduced hepatocyte ferroptosis by delivering miR-124-3p, which led to a significant decrease in the iron homeostasis factor, prostate six transmembrane epithelial antigen 3 [168]

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