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doi: 10.3390/nu15040812 44 Kalantar-ZadehK.JafarT

The increase in leu-enkephalin levels adds another anxiolytic dimension

DUBm mediates deubiquitination of H2B which in turn facilitates the recruitment of Ctk1

Physiological BCAA concentrations support anabolic processes and cellular repair, whereas chronic pathological elevation is associated with mTORC1 hyperactivation alongside impaired BCKDH-mediated catabolic flux, accumulation of branched-chain -keto acids (BCKAs) and branched-chain acylcarnitines, mitochondrial redox imbalance, and activation of cellular stress pathwayscollectively contributing to disrupted metabolic homeostasis, amplified inflammatory cascades, and mitochondrial dysfunction