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While the relevance of this finding to humans remains unclear, caution is advised in individuals with a personal or family history of medullary thyroid carcinoma or multiple endocrine neoplasia syndrome type 2

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GLP-1 therapies reduce activity in brain reward centers, which lowers cravings and constant thoughts about food

Mitochondria produce large amounts of ROS through the oxidation-reduction reactions carried out in the respiratory chain.2 Evidence indicates that under physiological conditions, ROS may participate in redox signalling, but excessive ROS production may cause oxidative damage to proteins, lipids and DNA, and affect cellular functionality.3 Several studies indicate the role of oxidative stress in the progression of autoimmune thyroid diseases, such as Graves disease.4 ROS are neutralised by the antioxidant enzymes SOD, CAT, and GPx