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Inhibition of brain CPT-1 decreases the entry of fatty acid carbons into the aqueous metabolic pools but does not alter the distribution into various long-chain acyl-CoA compounds and triglycerides
Current clinical evidence does not support an increased osteoporosis risk from GLP-1 medications themselves
Moreover, the role of endogenous glucagon-like peptide 1 (GLP-1) in the body is reviewed, including neuronally synthesized GLP-1, with a focus on how recent research developments may point towards therapeutic utility of GLP-1RAs in neuro-ophthalmology
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