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Early pharmacologic strategies primarily targeted isolated components of metabolic dysregulation, such as enhancing insulin secretion, reducing hepatic glucose output, slowing intestinal glucose absorption, or suppressing appetite

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Interestingly in PD patients, it has been shown, an specific increase in SOD levels in the substantia nigra, with no changes in activities of glutathione peroxidase, catalase and glutathione reductase (EC 1.8.1.7) [25]