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[2] This stability may support sustained biological activity across laboratory settings in which many shorter synthetic peptides would undergo rapid proteolytic degradation

As research interest around copper peptide research has accelerated through the 2020s, it has emerged as a foundational molecule for scientists investigating tissue remodeling and age-related decline

We found that wild-type (WT) ACTL6A and the DNBL, CC, and NBC ACTL6A mutants reversed the inhibition of cell proliferation and the reduced GCLC protein expression induced by knocking down ACTL6A expression, but the ACTL6A HR-deleted mutant did not show this restorative effect (Fig